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Kidney damage from diabetes is called diabetic nephropathy. It begins long before you have symptoms. An early sign of it is small amounts of protein in your urine. A urine test can detect it. A blood test can also help determine how well your kidneys are working. The earliest sign of diabetic kidney disease is an increased excretion of albumin in the urine. This is present long before the usual tests done in your doctor's office show evidence of kidney disease, so it is important for you to have this test on a yearly basis. Weight gain and ankle swelling may occur. You will use the bathroom more at night. Your blood pressure may get too high. As a person with diabetes, you should have your blood, urine and blood pressure checked at least once a year.This is the best wat for managing and preventing from diabetic diseases and complication caused due to it.
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Scope and Importance:
Diabetes and kidney disease Conference provides the scope for opportunities to learn progressed by international scientists and academicians. Diabetes and kidney disease Conference offers excessive quality content to suit the diverse professional development of these medicines to treat people all over the globe. It is a perfect platform to discuss the current discoveries and developments in the field of Diabetes and Nephrology.
Diabetic nephropathy is the leading cause of chronic kidney disease in patients starting renal replacement therapy and is associated with increased cardiovascular mortality. Diabetic nephropathy is a progressive kidney disease caused by angiopathy of capillaries in the kidney glomeruli. It is characterized by nephrotic syndrome and diffuse glomerulosclerosis. It is due to longstanding diabetes mellitus, and is a prime indication for dialysis in many developed countries. It is classified as a microvascular complication of diabetes. Diabetic nephropathy is more prevalent among African Americans, Asians, and Native Americans than Caucasians. Among patients starting renal replacement therapy, the incidence of diabetic nephropathy doubled from the years 1991–2001. Fortunately, the rate of increase has slowed down, probably because of the adoption in clinical practice of several measures that contribute to the early diagnosis and prevention of diabetic nephropathy, which thereby decreases the progression of established renal disease. Kidney failure provoked by glomerulosclerosis leads to fluid filtration deficits and other disorders of kidney function. There is an increase in blood pressure (hypertension) and fluid retention in the body plus a reduced plasma oncotic pressure causing edema. Other complications may be arteriosclerosis of the renal artery and proteinuria. Throughout its early course, diabetic nephropathy has no symptoms. They develop in late stages and may be a result of excretion of high amounts of protein in the urine or due to renal failure.
Diabetes is the most common cause of kidney failure, accounting for nearly 44 percent of new cases. Even when diabetes is controlled, the disease can lead to CKD and kidney failure. Most people with diabetes do not develop CKD that is severe enough to progress to kidney failure. Nearly 24 million people in the United States have diabetes, and nearly 180,000 people are living with kidney failure as a result of diabetes. Each year in the United States, more than 100,000 people are diagnosed with kidney failure, a serious condition in which the kidneys fail to rid the body of wastes. Kidney failure is the final stage of chronic kidney disease
International symposium and workshops
Aug 22-24, 2016, Sao Paulo, Brazil
November 02-04, 2015 Atlanta, Georgia, USA
February 29-March 02, 2016 Philadelphia, USA
June 06-08, 2016 Dallas, USA
Dec 5-7, 2016, Baltimore, USA
Nov 14-16, 2016, Dubai, UAE
Oct 20-22, 2016, Dubai, UAE
September 29-October 1, 2016, Toronto, Canada
Oct 17-19, 2016, Kuala Lumpur, Malaysia
Aug 22-24, 2016, Kuala Lumpur, Malaysia
Aug 8-10, 2016, Birmingham, UK
July 14-16, 2016, Cologne, Germany
July 11-13, 2016, Brisbane, Australia
Feb 29-Mar 2, 2016, Philadelphia, USA
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This page was last updated on 12th Sep, 2015
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